I believe this to be a very lazy and superficial video.
I would need to repeat a lot of what I wrote in the long PDF I linked in the OP, but some of the key points that that guy misses:
- lipid build-up on the skin: Why does he not question where it comes from? It is known that the skin...
What hair loss meds was your uncle on before?
In the case of your uncle and your friend, their results can be fully explained by hopping on treatment for the first time. Your gym friend's situation was not so bad so min/finasteride can do that alone. Your uncle at age 72 has very low T and DHT levels...
Amazing work my man. Regardless of whether your hypothesis holds true or not, the important thing is that you came up with something yourself and even collected data to back it up. Very valuable!
I will have a more in-depth look at everything you wrote later but want to add three things:
1...
Do you have sources for supporting this? Why would tension on its own lead to inflammation and calcification? Yes, inside a muscle tension can lead to inflammation, but the issue are the hairs. Also, calcification is not explained by this.
On the other hand, there are literally dozens of...
Because tension alone does not explain two other things happening:
- fibrosis
- calcification
I'll extend what I said before:
Inflammation + tension = fibrotic tissue regeneration
Inflammation without tension = soft tissue regeneration (or if not soft at least much less fibrotic)
No...
Only partially genetic. Diet during childhood and adolescence plays a big role, maybe even the bigger one. (source 1, source 2)
Definitely not genetic: Myopia - A modern yet reversible disease
Partially genetic, partially not. (see for example here the examples of an Asian monk vs. a Native...
But you know what causes excess sebum production? High-carb/high-sugar or lack of physical exercise.
A list of just a few sources confirming this:
Sebaceous gland lipids by M. Picardo et al:
Source
Source
Source
Source
Source
As you can see, insulin/carb/sugar issues are the main...
It is a good question. The answer is: scalp tension.
The perhaps most important thing to understand about scalp tension (and in fact any tension on the dermis) is that it is the strongest factor (among three or four) in determining whether the body will recreate tissue in a fibrotic or...
Ah, not surprising! What I also found surprising was this post in the thread you linked:
I haven't really dug into topical application of magnesium oil - maybe it could achieve what diet and supplements cannot?
Inositol should totally help in slowing hair loss down. In fact there is a...
Sort of causal. The common root cause is insulin resistance. Insulin resistance leads to damages in the vasculature: both in the central vasculature (CVD) and in the peripheral capillaries in the scalp, leading to hair loss.
Check out the PDF I attached to this post. It is a long read but...
I think there are different mechanisms at play with estradiol:
- Scavenging T and thus preventing conversion of T to DHT. Though this is likely not because of E2 itself but aromatase. Aromatase and 5ar compete for T.
- Influencing subcutaneous fat storage. Fat cell injection seems to boost hair...
@User27041995 I can help with this riddle.
When the body recreates tissue (which was damaged or needs replacement for other reasons, e.g. due to chronic inflammatory processes) there are several factors which influence the decision whether new tissue is recreated fibrotic or non-fibrotic. The...
Have you even had a look at the PDF I posted in the opening post?
If what you say were true, then all genetically identical twins should be balding at the same speed. Have a look at pages 7 and 8. There I included photos from studies that compared the balding rates of monozygotic, so...
Long story short: Regrowth likely not possible. The only thing you can do is halt further hair loss progression.
How would you do this? By eliminating all forms of insulin resistance, both primary (diet/exercise) and secondary (sleep, stress, avoiding overexercise, and a few others).
Fermented...