My background: I’m 23 and Norwood 1,5. I have some diffuse thinning in crown area and behind hairline, but I don’t have experienced any hairline recession. I don’t know if anyone has noticed my thinning, because it is still in early stages.
Well, this my 6 months update. I’ve been on Proscar 1,25 mg/day for over six months. I added to my daily regimen 1 g MSM, 150 mg GSE, fish liver oil and B-vitamins in July.
Results: Well, the photos which I have taken with digital camera from April shows that I have gained no regrowth nor decreased hair count. I so guess I have maintained my hair during these six months. Still, I’m concerned about a couple of things.
1. First, a classic question: what is normal range of losing hair daily? I have lost these six months 100-150 terminal hairs in a day! That’s too much, or it this still normal for Norwood 1,5? Daily shed rate hasn’t decreased at all during these six months.
2. Second, many diffuse thinners use finasteride. But does it really help them? I found this study in July from pubmed:
"Production rates of testosterone and of DHT"
Testosterone may be more important than DHT in balding women.
Metabolism. 2003 Jul;52(7) 27-929. Related Articles, Links
Production rates of testosterone and of dihydrotestosterone in female pattern hair loss.
Vierhapper H, Maier H, Nowotny P, Waldhausl W.
Production rates of testosterone (T) and of dihydrotestosterone (DHT) were determined in young women (n = 8, age, 23 to 40 years) with female-pattern hair loss using the stable isotope dilution technique and mass spectrometry. 1alpha,2alpha-d-testosterone and 2,3,4-(13)C-dihydro-testosterone were infused for 10 hours at a dose of 2 microg/h each and blood samples were obtained at 20-minute intervals during the last 4 hours of the observation period. In the presence of normal metabolic clearance rates (MCRs), production rates of T were increased (9.4 +/- 5.0 microg/h; normal, 4.3 +/- 1.9 microg/h, P <.05). MCRs of DHT (8.0 +/- 3.4 L/h; normal, 25.9 +/- 12.3 L/h, P <.002) were subnormal in all women and the production rates of DHT were within or below the normal range (mean, 1.6 +/- 0.6 microg/h; normal, 2.9 +/- 1.1 microg/h, P <.02). Unlike men with male-pattern baldness, women with female-pattern baldness are characterized by increased production rates of T, but not of DHT. These results are compatible with the idea that 5alpha-reductase inhibition is of no therapeutical value in female-pattern baldness.
PMID: 12870172
This article got me concerned. Does this mean that diffuse type of loss in women and in MEN is caused by testosterone, not DHT? Are the fpb and â€diffuse-type†of male pattern baldness the same thing?
Well, this my 6 months update. I’ve been on Proscar 1,25 mg/day for over six months. I added to my daily regimen 1 g MSM, 150 mg GSE, fish liver oil and B-vitamins in July.
Results: Well, the photos which I have taken with digital camera from April shows that I have gained no regrowth nor decreased hair count. I so guess I have maintained my hair during these six months. Still, I’m concerned about a couple of things.
1. First, a classic question: what is normal range of losing hair daily? I have lost these six months 100-150 terminal hairs in a day! That’s too much, or it this still normal for Norwood 1,5? Daily shed rate hasn’t decreased at all during these six months.
2. Second, many diffuse thinners use finasteride. But does it really help them? I found this study in July from pubmed:
"Production rates of testosterone and of DHT"
Testosterone may be more important than DHT in balding women.
Metabolism. 2003 Jul;52(7) 27-929. Related Articles, Links
Production rates of testosterone and of dihydrotestosterone in female pattern hair loss.
Vierhapper H, Maier H, Nowotny P, Waldhausl W.
Production rates of testosterone (T) and of dihydrotestosterone (DHT) were determined in young women (n = 8, age, 23 to 40 years) with female-pattern hair loss using the stable isotope dilution technique and mass spectrometry. 1alpha,2alpha-d-testosterone and 2,3,4-(13)C-dihydro-testosterone were infused for 10 hours at a dose of 2 microg/h each and blood samples were obtained at 20-minute intervals during the last 4 hours of the observation period. In the presence of normal metabolic clearance rates (MCRs), production rates of T were increased (9.4 +/- 5.0 microg/h; normal, 4.3 +/- 1.9 microg/h, P <.05). MCRs of DHT (8.0 +/- 3.4 L/h; normal, 25.9 +/- 12.3 L/h, P <.002) were subnormal in all women and the production rates of DHT were within or below the normal range (mean, 1.6 +/- 0.6 microg/h; normal, 2.9 +/- 1.1 microg/h, P <.02). Unlike men with male-pattern baldness, women with female-pattern baldness are characterized by increased production rates of T, but not of DHT. These results are compatible with the idea that 5alpha-reductase inhibition is of no therapeutical value in female-pattern baldness.
PMID: 12870172
This article got me concerned. Does this mean that diffuse type of loss in women and in MEN is caused by testosterone, not DHT? Are the fpb and â€diffuse-type†of male pattern baldness the same thing?
