most common reported sleep order under finasteride seems to be that you cant mantain sleep and always wake up earlier...
its similar to sleeping problems parkinson patients report .... and we know there a similarities between parkinson and finasterid regarding the effecr on the brain - both f*** with your dopamin in the brain
"Unlike many older adults, patients with Parkinson’s disease often find that they have no trouble initiating sleep, but often wake up within a few hours and find sleeping through the rest of the night to be difficult."
http://pdcenter.neurology.ucsf.edu/patients-guide/sleep-and-parkinsons-disease
"Sudden Decline in Testosterone May Cause Parkinson’s Disease Symptoms in Men
While scientists use different toxins and a number of complex genetic approaches to model Parkinson’s disease in mice, we have found that the sudden drop in the levels of testosterone following castration is sufficient to cause persistent Parkinson’s like pathology and symptoms in male mice,” said Dr. Kalipada Pahan, lead author of the study and the Floyd A. Davis endowed professor of neurology at Rush. “We found that the supplementation of testosterone in the form of 5-alpha dihydrotestosterone (DHT) pellets reverses Parkinson’s pathology in male mice.”"
https://www.rush.edu/news/press-rel...one-may-cause-parkinsons-disease-symptoms-men
what is very disturbing that in some cases these sidw effects of finasteride stay even after quiting or even just occur first after quiting. prinably due to permanent damage of the receptors in the brain
"
A growing body of research is offering clues about why finasteride might cause these symptoms in a subset of men. It suggests the enzyme that Propecia shuts down—5-alpha-reductase—does a lot more than convert testosterone to the hair-killing dihydrotestosterone (DHT). It also converts hormones like cortisol and progesterone into "neurosteroids" that play key roles in the brain.
"One recent study published in
Neuroendocrinology found that after 20 days of finasteride exposure, male rats showed altered levels of a whole host of neurosteroids and receptors in their brains. Thirty days after going off the drug, the changes were
more pronounced than when they were on the drug.
Studies in humans have only just begun, at Baylor, Boston University, and elsewhere, so it's way too early to draw firm conclusions. But the theory surrounding what some patients call "the crash" goes something like this: Cells in the brain and genitals are starved of important hormones while on the drug, so they grow more receptors to sop up all that they can get. Once the drug is discontinued, the hormones come flooding back with more than the cells can handle, which hurts or kills them. "Essentially, the cells get too much DHT, it puts them in overdrive and it burns them out," says Jacobs. So even if the body starts making all those missing compounds again, the tissue has trouble using them"
https://tonic.vice.com/en_us/articl...y-behind-americas-best-selling-hair-loss-drug