finasteride substitue

[helpwithhair]

How does the following sound as a reasonablely good replacement for finasteride

saw palmetto
beta sitosterol
green tea extracts
He Shou Wu
soy isoflavone
Pumpkin Extract

taken daily

 

[TheGrayMan2001]

None of them replace finasteride.

Herbal supplements are scientifically proven to work. Studies have been done with few to no results with most of that. The rest is psuedoscience crap that people want to believe because they don't like the idea of finasteride.

If you have not tried finasteride, you must try it. You will probably be fine and have no side effects.

 

[hairrific]

finasteride will halt the hair loss for you, yes, thank god for finasteride. But lets not forget that I think it is easy to just pop a Rx and not look any further and I think that is a safe, cozy feeling, mass mega culture rut we get in sometimes, so here is a finasteride substitute maybe:

Abstract
Background

Maintaining endogenous testosterone (T) levels as men age may slow the symptoms of sarcopenia, andropause and decline in physical performance. Drugs inhibiting the enzyme 5?-reductase (5AR) produce increased blood levels of T and decreased levels of dihydrotestosterone (DHT). However, symptoms of gynecomastia have been reported due to the aromatase (AER) enzyme converting excess T to estradiol (ES). The carotenoid astaxanthin (AX) from Haematococcus pluvialis, Saw Palmetto berry lipid extract (SPLE) from Serenoa repens and the precise combination of these dietary supplements, Alphastat® (Mytosterone(™)), have been reported to have inhibitory effects on both 5AR and AER in-vitro. Concomitant regulation of both enzymes in-vivo would cause DHT and ES blood levels to decrease and T levels to increase. The purpose of this clinical study was to determine if patented Alphastat® (Mytosterone(™)) could produce these effects in a dose dependent manner.

Conclusion

The precise combination of AX and SPLE, Alphastat® (Mytosterone(™)), produced significant changes in serum T, DHT and ES levels. A dose of either 800 mg or 2000 mg/day produced significant increases in T and decreases in DHT within three days with no increases in ES. The effect was not dose dependent indicating that the 800 mg/per day dose is as equally effective as 2000 mg/day in the age range of subjects studied. Blood levels of ES also decreased significantly and in a dose dependant manner indicating the 2000 mg/day dose is more effective than the 800 mg/day dose. There were no outward signs of toxicity or adverse reactions. This data provides support for each mechanism of action observed in-vitro and suggests a potential role for its use in aging men experiencing TDS or symptoms of BPH.

http://www.jissn.com/content/5/1/12