Dr. Wongs article

[pegasus2]

If T converts into DHT how would lower total/free T increase the rate of that conversion into DHT? It makes no sense. Having lower T levels doesnt really mean anything because its sensitivity to androgens that causes male pattern baldness not levels of hormones. Your just more likely to keep your hair by lowering DHT since its all we know how to do so far.

It doesn't take much T to make DHT. If T is low your body needs to convert it to the more potent DHT to compensate.

Androgen sensitivity isn't everything. If you had no androgens it wouldn't matter how sensitive your follicles are to them. Hormone levels do matter, that's why finasteride works for most people.

 

[Afro_Vacancy]

It doesn't take much T to make DHT. If T is low your body needs to convert it to the more potent DHT to compensate.

Androgen sensitivity isn't everything. If you had no androgens it wouldn't matter how sensitive your follicles are to them. Hormone levels do matter, that's why finasteride works for most people.

To the best of my recollection there are no strong correlations between serum androgen levels and male pattern baldness.

 

[atthekeysbored]

Yes high estrogen and low testosterone causes men to bald, that is why women are more bald than men and why men who transition using estrogen related products lose all their hair permenantly from the sudden estrogen spike.
Pure bro science b.s.

 

[BTW]

To the best of my recollection there are no strong correlations between serum androgen levels and male pattern baldness.

after numerous hours spent reading everything, the only conclusion I can come to is that follicles sensitivity to androgens is an inherited QUANTITIVE, not calitative trait
You can make the same analogy as height, some are taller, some are smaller. In the same way some people are more sensitive, others are more resistant. The DNA code for some of us is more defective, after a certain number of life cycles it stop functioning as it should. The time at which it does this is very clear, for others it's in their 20s, for other in the 50s. An autistic researcher can probably count the DNA codons and tell you exactly when your hair will decide to miniaturize.
https://en.wikipedia.org/wiki/Telomere
"The telomeres are disposable buffers at the ends of chromosomes which are truncated during cell division; their presence protects the genes before them on the chromosome from being truncated instead."
"The telomeres are disposable buffers"
"disposable buffers"

after a certain number of divisions something similar happens to our hair follicles. Some have disposable buffers that last a life-time, some get the shitty deal.

I'm looking at my father, I inherited his exact balding pattern, at the exact age. Our living conditions, early life were totally diferent, food, lifestyle choices, you can name it. Yet we ended up with the same hair loss, I know because I saw pictures of him at my age, we have the exact hair pattern loss. One funny thing is how does the body know how to distribute these defective follicles right at the crown and the temple areas. The shitiest follicles are designated to this area, what does it actually mean? The worst follicles don't end up in the occipital regions or behind your ears, always starting at the temple region in a very deliberate pattern.

Male pattern baldness is present in females and in males alike. Our sisters have our balding follicles but the androgens are not there for them. Unlucky females with defects in the metabolism of adrenal hormones end up with virilization and experience MBP in their 20s just like us.