I would hold off on saying seti is purely just maintenance until the trials being done are published later this year. Think of this: Propecia in the end has the same effect of seti but does it in a different way. Propecia blocks conversion of DHT, less DHT attaches to follicle so then downstream there is less PGD2 which attacks the follicle. Seti works by competing with PGD2 at the receptor so there is less PGD2 attacking the follicle. In the end both result in less PGD2 hurting the follicle.
It's an established fact that for Propecia results are completely different for everyone. Some get maintenance, some get a little regrowth, some get crazy regrowth, and some don't get anything at all. Since as I said before in the end both end up reducing PGD2 I can't see why seti couldnt have a range of results for users like propecia does.