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Can you explain why you use metformin?
Also I would say for folks to look for changes in skin in general as a possible launching point for at least some re-growth since hair and skin are closely related.
Exploring The Hormonal Route. Hair=life.
- Thread starter bridgeburn
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Also I would say for folks to look for changes in skin in general as a possible launching point for at least some re-growth since hair and skin are closely related.
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May I ask, how was your oxidative stressed measured?
And do you know the root cause of your oxidative stress (induced by high glucose etc)?
Resveratrol not only lowers adrenal androgens but might also be beneficial for oxidative stress.
Role of resveratrol in regulation of cellular defense systems against oxidative stress - PubMed
Resveratrol, a natural polyphenolic compound, is found in various kinds of fruits, plants, and their commercial products such as red wine. It has been demonstrated to exhibit a variety of health-promoting effects including prevention and/or treatment of cardiovascular diseases, inflammation...
pubmed.ncbi.nlm.nih.gov
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Nobody here is doing science. And I don't mean that perjoratively, this is just simply not what science is. If we had millions of dollars maybe we could actually develop a hair growth plan that works for most people, but the truth is right now everyone here is just feeling around in the dark hoping to maximize their results. I think it's been proven that altering hormones can improve hair, however there is no formula and no matter how much discussion we have on this forum we will never arrive at a formula. We can come up with speculation but really we'll never have anything more.
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Я проведу эксперемент,я на протяжении месяца не буду есть ничего подавляюще сахарного,это кофе 3-1,сахар,напитки газированные,конфеты,печенье,кока кола,сладкие чаи,сладкие булки,сгущенка,паста шоколаднаяВсе что содержит в себе много сахара.У меня будет обычная еда!
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All true. It's just that it's chaotic for even semi-outsiders to understand. With @bridgeburn, you had essentially one main voice articulating and this person was on an extended HRT formula plus oral min that was pretty easy to follow. At times, he would change AA's or go off AA's but his protocol appeared to be one that shot for well above adult female levels.
A lot of this is exactly the same with the topical use of estrogen on the scalp and which makes me shy away from categorical statements about Estrogel while using it in basically unlimited amounts of scalp and other places. It might just be the climara patches. I take some premarin, cheap and reputed to be extra good for breast modeling. Perhaps it works better for hair too but who knows?
So all's I knows is that everywhere I slathered Estrogel, the area got big, in terms of below the neck usage. The two main examples are breasts and incipient female waist/hips towards pear shape. I put it on my face and my face looked better and younger. I tried it on my hair and I experienced a period of increasing hair regrowth. Maybe all of these things would have happened just from oral E2 or a higher dosage of premarin but for me, they were all associated with direct and copious topical estrogen use. But if someone XY just pm's me, I stammer a bit as to whether estrogen topicals matter at all or if they even work. That's because we don't have the science to back up my anecdotal assertions but I have seen substantial changes even from scrotal application which is recommended for many MtF's now and the entire area appears to be far more estrogenized and intersex in aspect. My foreskin grew back, at least a huge amount of it but it resembles more a clitoral hood. The entire area is fleshy and plump and it never was like this prior to HRT.
So the changes on genitalia can be substantial, even more substantial than many imagine they would be on the penis. The penis loses very little actual size but it starts behaving like a clitoris as do the scrotum. How do I know? Well, the tissue begins to move and structure itself differently. The penis is no longer anchored but basically is along for the ride whenever the "mound" is manipulated or moved. There's a pseudo-opening and pseudo-mons. Only stereotypical female methods of masturbation work like vibration or direct pressure against something like my teddy bear. Up and down movements do nothing.
Now I am on mega-amounts of hormones but if anything the questions about will one's dick or nut shrinkage are off-target per se. The issue is more so whether they will substantially feminize then any size differences. The penis will develop a tendency to be more so internal but if you yank on it, the length varies little. If you hold the entire area down until the pubic bone, which anchors the penis, losses of fifty percent of length are possible. Remember, penile tissue equals labia minora tissue and scrotal equals labia majora tissue, and these areas may begin behaving much more like the XX equivalents. So, by putting pressure on the mound-like area below the penis/clitoris, said organ will retrench into what now acts as a clitoral hood and be more anchored. Below that, one can press quite far into the indentation and the same sorts of female stimuli work, meaning things that push the scrotal/labia majora tissue apart as much as possible. A phallic-shaped item can be used roughly equivalently to cis-female phallus use, even though there is no actual opening.
So while I have been dismissive of the penis shrinking questions, I believe that the potential changes to XY genitalia from MtF HRT can be exceedingly profound to the point that surgery might seem superfluous as everything changes and inverts.
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It's most likely due to my lifestyle and psychological issues (stress). I dunno how the Doctor measured it, but he showed me clear pictures of my blood cells and they were actually dying.May I ask, how was your oxidative stressed measured?
And do you know the root cause of your oxidative stress (induced by high glucose etc)?
Resveratrol not only lowers adrenal androgens but might also be beneficial for oxidative stress.
Role of resveratrol in regulation of cellular defense systems against oxidative stress - PubMed
Resveratrol, a natural polyphenolic compound, is found in various kinds of fruits, plants, and their commercial products such as red wine. It has been demonstrated to exhibit a variety of health-promoting effects including prevention and/or treatment of cardiovascular diseases, inflammation...
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I am also using Metformin, but for different reasons. Good to know that it is also beneficial for hair. Thanks!
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Сейчас я беру эстрадиол в геле и прогестерон.В зонах андрогенной алопеции у меня выросли маленькие волосики,они никак не могут набрать длинну
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Now that I have a plan in mind: Bica + E2 4mg for the first three months and then E2 6mg^ monotherapy. Now I just want to figure out if progesterone would be a helpful addition later on down the road or if it'd just be superfluous.
Given that my hairloss is genetic, it may just be run of the mill male pattern baldness, the kind of hairloss that could be more surely helped by HRT. But, just to be sure, I will start directing my research into dealing with the backdoor passage ways and how to deal with them. Just in case.
Also, there's the business of explaining this all to the doctor I get.
My goal will be to achieve longer and stronger hair growth, but at the minimum, I'd be fine with having hair long and strong enough to support extensions and various braided styles.
Given that my hairloss is genetic, it may just be run of the mill male pattern baldness, the kind of hairloss that could be more surely helped by HRT. But, just to be sure, I will start directing my research into dealing with the backdoor passage ways and how to deal with them. Just in case.
Also, there's the business of explaining this all to the doctor I get.
My goal will be to achieve longer and stronger hair growth, but at the minimum, I'd be fine with having hair long and strong enough to support extensions and various braided styles.
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Why do you want to start with bica and then drop it? You can just start at 6mg E. Progesterone is good, it suppresses T production and also competes with AR-receptor and also directly with T for 5a-reductase IIRC.
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I have some reservations about how effective monotherapy is.
So, the Bica is to give the E2 an early advantage to take hold against T/DHT.