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The mechanism by which they raise T is entirely different though? Where's the puzzle supposed to be in this?
Exploring The Hormonal Route. Hair=life.
- Thread starter bridgeburn
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Isn’t there studies done with bic and avodart used in conjunction as a more aggressive cancer treatment to either being use alone?
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So if we can’t use it with bic how do we block dht while using bic
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finasteride will still block 70% of your dht. cpa if you are brave enough.
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hello Janey, what was that store called which transgender folks order stuff online - I believe it was in Canada? You mentioned it on your other thread. Just too pressed for time these days to look through everything.
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I believe it was kennedy that mentioned taking bica with dutasteride increases level of dutasteride significally in the blood - isn't that a good thing? Maybe you can even taper dutasteride to 2-3 times a week and that would balance just right.
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No, dutasteride in your blood implies it's not being properly processed by your liver.
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HRT.cafe has a long list but I think I am not supposed to mention any except in private messages.
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I think that the growing consensus is that dutasteride is for life even after SRS DHT can pop back up with virtually no T in my blood and cause a tiny bit of dermatitis and most of that stopped with beard removal until oral min brought it back but I can taper down at some point. I continue to think that it is optimal to well exceed the standard norms for levels of e2, which aren't all that high. You have to blow the doors out to get rid of DHT and then it sometimes still comes back. DHT basically ruined my life by causing the conflict with my hair fall increasing and my constant dermatitis. I couldn't shave without bleeding and it took me an extraordinary long time to shave because of the bleeding and infection. All but entirely gone under the regime of Goddess.
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The brazen-ness of youth. You can't promise someone that, can you? I guess I do it sometimes too when I am positive about something.
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But why would finasteride work when dutasteride didn't? That's actually a key question for me because I was taking both and figured it was overkill although covered. Most of my meds and all of my surgeries will be covered by my insurance so I would almost have to be crazy not to get free SRS since it normally costs like a million dollars out of pocket. They have sweetened the deal so much and I just can't pass up a bargain now that I am blonde-ish.
Do you disagree on something? Of course, no one can guarantee that this will help. For some, even 150mg of Bicalutamide does not help; there are many variables beyond our control. However, Finasteride does not conflict with Bicalutamide and is not as harmful to the liver as CPA + Bicalutamide. Also, Bicalutamide does most of the work. Dutasteride is financially questionable
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This is my thought exactly? Why does one work and not the other?
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Marky! Hiya. Long-time no see.....
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Different chemical structure? Just because the effects are comparable doesn't mean the responsible chemical structure is.
Good point. Maybe it has to do with the interactions of its metabolites.
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Well, according to James Booker, anyone who uses both finasteride and dutasteride is an idiot because they both do the same thing which is rape your system of the hormones that you need to function.
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I know why in general one is preferable to the other. Finasteride especially titrated probably doesn't work as well as dutasteride and therefore might be less prone to cause sheds. It's the same thing with 2 percent min. Now why do they sell inferior min for the same price or whatever? It's because two percent is less likely to cause sheds especially among women but really anybody should pretty much always start low and titrate upwards. I was always like, nah, I need five, ten, 12 and even 15 percent min. I never shed on this med but apparently many do because they start at 5 percent. Same thing with finasteride. For guys, there might be some benefit to removing less, not more of certain types of reductases if they aren't relevant to scalp DHT. Prostate and HRT meds tend to overlap especially non-structurally estrogen-based molecules like spironolactone and CPA and MPA and now bica and previously dutasteride and proscar.