My Zix Experience - How B6 And Zinc Have Saved My Hair

sonictemples

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The whole cetirizine thing was abandoned because some people weren't able to grasp the concept of prostaglandins. It's not that important how much PGD2 you have inhibited but what matters is how the prostaglandin ratio on the scalp tissue has changed.

Diclofenac in this case is little bit of a gamble since it works on COX inhibition and not on mast cell regulation like cetirizine. I would have some sort of concept on how I could increase PGE2 artificially (minoxidil, oral castor oil, ricinoleic acid, caffeine ginseng taurine and the list goes on) prior to thinking about messing with the COX inhibition.

I have seen people losing hair on caffeine, indomethacin (was considered a cure a while back) and other NSAIDs like aspirin.

Because they all failed to take PGE2 into consideration. Once people think something doesn't work as well, they tend to ditch it instead of looking at the treatment in several angles.

Once you grasp a concept well, you can add that to your well-grounded regimen to see more halt in hair loss or even regrowth.

In my experience PGD2 limits the vellus hairs that could've popped up even if someone is already on a well-supported treatment regime. It's up to you too increase the PGE2 levels to possibly thicken those velluses.

So, yeah, most people here a while back never had much of an understanding nor patience to evaluate and revise their plans.

PGD2 studies were barely coming out and they pretty much took a shot in the absolute dark and obviously didn't achieve what they wanted.

This is even the case for Allergan, they dropped setipiprant like nothing lol, same for Novartis.
 
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inmyhead

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The whole cetirizine thing was abandoned because some people weren't able to grasp the concept of prostaglandins. It's not that important how much PGD2 you have inhibited but what matters is how the prostaglandin ratio on the scalp tissue has changed.

Diclofenac in this case is little bit of a gamble since it works on COX inhibition and not on mast cell regulation like cetirizine. I would have some sort of concept on how I could increase PGE2 artificially (minoxidil, oral castor oil, ricinoleic acid, caffeine ginseng taurine and the list goes on) prior to thinking about messing with the COX inhibition.

I have seen people losing hair on caffeine, indomethacin (was considered a cure a while back) and other NSAIDs like aspirin.

Because they all failed to take PGE2 into consideration. Once people think something doesn't work as well, they tend to ditch it instead of looking at the treatment in several angles.

Once you grasp a concept well, you can add that to your well-grounded regimen to see more halt in hair loss or even regrowth.

In my experience PGD2 limits the vellus hairs that could've popped up even if someone is already on a well-supported treatment regime. It's up to you too increase the PGE2 levels to possibly thicken those velluses.

So, yeah, most people here a while back never had much of an understanding nor patience to evaluate and revise their plans.

PGD2 studies were barely coming out and they pretty much took a shot in the absolute dark and obviously didn't achieve what they wanted.

This is even the case for Allergan, they dropped setipiprant like nothing lol, same for Novartis.

Well they dropped it, because it has shown no results for hair loss. Why do you think they are wrong? And how can you know better than official studies?
 

sonictemples

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Well they dropped it, because it has shown no results for hair loss. Why do you think they are wrong? And how can you know better than official studies?

I have seen people witnessing success while it was used with certain compounds that are really good for PGE2. This is the problem of hair loss companies, they do not care about us but our money, that’s all.

Follica isn’t cure, this isn’t cure that isn’t cure... What if we combined them all? And we see people do it here all the time. That doesn’t mean that they know less, they are the ones creating the compounds after all. They drive motivation from money, we from hair. No company would touch their hands on for example Zix, and Cetirizine? Why? Are they BS? Of course not, they cannot spend that much money on something they cannot possibly patent. In the case of Allergan, they had pretty nice compounds that could be used to increase PGE2, but they were expensive so they ditched it. Imagine that with setipiprant, and it would be a good treatment for some.

That out of the way, let’s turn back to Zix as this post intends. If you have the urge to discuss, you are always welcome in my PMs, have a good one!
 

db135

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killDHT

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The whole cetirizine thing was abandoned because some people weren't able to grasp the concept of prostaglandins. It's not that important how much PGD2 you have inhibited but what matters is how the prostaglandin ratio on the scalp tissue has changed.

Diclofenac in this case is little bit of a gamble since it works on COX inhibition and not on mast cell regulation like cetirizine. I would have some sort of concept on how I could increase PGE2 artificially (minoxidil, oral castor oil, ricinoleic acid, caffeine ginseng taurine and the list goes on) prior to thinking about messing with the COX inhibition.

I have seen people losing hair on caffeine, indomethacin (was considered a cure a while back) and other NSAIDs like aspirin.

Because they all failed to take PGE2 into consideration. Once people think something doesn't work as well, they tend to ditch it instead of looking at the treatment in several angles.

Once you grasp a concept well, you can add that to your well-grounded regimen to see more halt in hair loss or even regrowth.

In my experience PGD2 limits the vellus hairs that could've popped up even if someone is already on a well-supported treatment regime. It's up to you too increase the PGE2 levels to possibly thicken those velluses.

So, yeah, most people here a while back never had much of an understanding nor patience to evaluate and revise their plans.

PGD2 studies were barely coming out and they pretty much took a shot in the absolute dark and obviously didn't achieve what they wanted.

This is even the case for Allergan, they dropped setipiprant like nothing lol, same for Novartis.
I do not think seti is rubbish.Someone said it could wolk with oral 4g one day.
If I remember correctly, the SETI used in that company's experiment is no more than 2G per day. This may be why the company didn't see the results. I still remember someone combining SETI external use with acupuncture and minoxidil. He said that when he stopped using SETI external use, he lost more. One more time and it's back. It is not through PGD2 that there is no result, many people have achieved the successful result of reducing PGD2 in the forum.
 

sonictemples

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Also, one product called Crinagen is pretty much took the Azelaic-Zinc-B6 study as its core. However, they have used Zinc Acetate Hydrate instead of the zinc that we use. I will try to contact some chemists there to ask about the idea behind this preference.
 

sonictemples

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The spray has pretty good reviews than other natural treatments. The study may be a rare diamond.
 

killDHT

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After using micro needle.I use cet.And I use Diclofenac to kill all the pgd2 48 hours later.I only need two days of PGE2. And two days of zero PGD2
 

sonictemples

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I am using just a regular alcohol from the pharmacy
Is it ok?

Knowing that the reason behind the usage of distilled water specifically ia to decrease or minimize the chances of zinc getting distrupted by the ions from the water, hence why we add distilled first, and then alcohol, it shouldn’t matter too much.
 

Stating facts

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I have that one but its not the vegan version. Im not using alcohol but has not worked for me as far as itch or shedding

Is this heptahydrate?

On this bottle, they write that "Long term intake may result to anaemia", does it also apply to topicals as well? Is anyone aware behind the science here?
 

sonictemples

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On this bottle, they write that "Long term intake may result to anaemia", does it also apply to topicals as well? Is anyone aware behind the science here?
On this bottle, they write that "Long term intake may result to anaemia", does it also apply to topicals as well? Is anyone aware behind the science here?


Too much zinc, taken in the form of dietary supplements, may disrupt copper uptake, leading to neurological problems and anemia, the evidence indicates. ... Low levels of albumin (below 25 mg/l) are linked to low zinclevels as are high (above 20 mg/l) levels of CRP.
 

db135

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Is any one else dermastamping with zix? Im not having any results but have seen other people with great results with original zix elsewhere combined with needling.

Do you apply zix straight after needling?

I bought a 1mm stamp and was going to use it every 4 days combined with original zix.

But again i dont have the same zinc as the op uses

Thanks
 

sonictemples

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Is any one else dermastamping with zix? Im not having any results but have seen other people with great results with original zix elsewhere combined with needling.

Do you apply zix straight after needling?

I bought a 1mm stamp and was going to use it every 4 days combined with original zix.

But again i dont have the same zinc as the op uses

Thanks

I do 3mm right after it
 

sonictemples

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Are you not worried about hitting the hair follicle?

How often are you doing that? How are you results?

I also use caffeine with it. It pretty much halted my hair loss but loses its efficacy after a while.
 

sonictemples

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A good scalp biopsy should include terminal hair bulbs and usually is 0.8 to 1cm deep.
 

sonictemples

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