Worried about gyno !
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aRaPoRn
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i have now been on propecia for 2 months the tissue on my chest has worsened but i have gained more muscle, when i tense my muscles you can easily feel a fair amount of "tissue" im worried about this fees really wierd, if it really is gyno, the glandular type if i stopped finasteride would it go\??
hair mchair
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Long time no see Telo. How's it hangin?
TEST SUBJECT#1
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Gyno can consist of three parts: gland alone, mixed gland and fat and fat alone. If a person is only increasing size in the chest area and no where else on the body chances are good its gyno.
You dont have to feel any hard lumps or bumps to have gyno if its fat. If the gland itself has grown, it WILL be felt as a pea sized lump behind the nipple area. Gyno can reach all the way back and under the arm. If your nipples are sore to sqeeze, if they itch or are tender to the touch, chances are good that you have gyno. Since every human body is different it would be foolish to believe that Propecia could not effect someone extremely sensitive to hormonal changes in a VERY SHORT period of time.
If you are new to Propecia and have expereinced chest sesnitivity do NOT ignore it if you are worried about growing boobs.
Gyno WILL NOT go away on its own if left untreated. Obviously pervention is the word to remember, anti estrogens help with prevention of gyno. All is not lost if a person does get gyno, as it is easily fixed with the help of an experienced surgeon.
Hope this helps.
You dont have to feel any hard lumps or bumps to have gyno if its fat. If the gland itself has grown, it WILL be felt as a pea sized lump behind the nipple area. Gyno can reach all the way back and under the arm. If your nipples are sore to sqeeze, if they itch or are tender to the touch, chances are good that you have gyno. Since every human body is different it would be foolish to believe that Propecia could not effect someone extremely sensitive to hormonal changes in a VERY SHORT period of time.
If you are new to Propecia and have expereinced chest sesnitivity do NOT ignore it if you are worried about growing boobs.
Gyno WILL NOT go away on its own if left untreated. Obviously pervention is the word to remember, anti estrogens help with prevention of gyno. All is not lost if a person does get gyno, as it is easily fixed with the help of an experienced surgeon.
Hope this helps.
TEST SUBJECT#1
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Talogen what state you live in? (Just assuming you live in the states.)
TEST SUBJECT#1
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I read of a very good doctor who does operations for bodybuilders who frequently get gyno because of huge steroid cycles. He works on all of them so he obviously has a great deal of knowledge on the subject. i will try to get you the name later today, right now too busy.
G
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Finasteride use (at 5MG) and side effects
Interesting side effect rates at the 5MG dosage.
See below
Finasteride in the treatment of benign prostatic hypertrophy: an update. New indications for finasteride therapy.
Ekman P.
Department of Urology, Karolinska Hospital, Stockholm, Sweden.
A phenomenon of the prostate gland, which is also shared by hair follicles, is that it is little influenced by testosterone (T) for androgenic stimulation, but instead by its metabolite 5alpha-dihydrotestosterone (DHT). By blocking the conversion of T to DHT, the circulating level of DHT is reduced by 80%, the size of the prostate gland is reduced by about 20% and the level of prostate-specific antigen (PSA) by about 50%. Treatment of patients with obstructive benign prostatic hypertrophy (BPH) with the drug Finasteride leads to a moderately improved urinary flow, symptomatic improvement and halts the natural progress of the disease. Since DHT potentiates the effect of testosterone on erectile function, the side-effects are impotence in 3% of patients, decreased ejaculatory volume, and gynaecomastia in 0.4% of patients. The drug could be regarded as a safe way to treat moderately symptomatic BPH. The efficacy of the drug is long-lasting (more than 7 years). It has also been tried in prostate cancer, but is less effective. It reduces PSA levels by 50% and, in combination therapy, therefore, PSA levels remain low for longer when Finasteride is added. An important finding is the efficacy of Finasteride treatment in haematuria from BPH. The drug interacts with vascular endothelium growth factor and efficiently prevents new bleeding. It could be regarded as a first-line therapy for this type of haematuria. Finasteride can also be used to stop male baldness. It seems particularly effective in men aged 20-40 years; 85% of patients stopped losing hair when given Finasteride. When the treatment was stopped hair loss continued, thus therapy may have to be "lifelong".
Publication Types:
Review
Review, Tutorial
Interesting side effect rates at the 5MG dosage.
See below
Finasteride in the treatment of benign prostatic hypertrophy: an update. New indications for finasteride therapy.
Ekman P.
Department of Urology, Karolinska Hospital, Stockholm, Sweden.
A phenomenon of the prostate gland, which is also shared by hair follicles, is that it is little influenced by testosterone (T) for androgenic stimulation, but instead by its metabolite 5alpha-dihydrotestosterone (DHT). By blocking the conversion of T to DHT, the circulating level of DHT is reduced by 80%, the size of the prostate gland is reduced by about 20% and the level of prostate-specific antigen (PSA) by about 50%. Treatment of patients with obstructive benign prostatic hypertrophy (BPH) with the drug Finasteride leads to a moderately improved urinary flow, symptomatic improvement and halts the natural progress of the disease. Since DHT potentiates the effect of testosterone on erectile function, the side-effects are impotence in 3% of patients, decreased ejaculatory volume, and gynaecomastia in 0.4% of patients. The drug could be regarded as a safe way to treat moderately symptomatic BPH. The efficacy of the drug is long-lasting (more than 7 years). It has also been tried in prostate cancer, but is less effective. It reduces PSA levels by 50% and, in combination therapy, therefore, PSA levels remain low for longer when Finasteride is added. An important finding is the efficacy of Finasteride treatment in haematuria from BPH. The drug interacts with vascular endothelium growth factor and efficiently prevents new bleeding. It could be regarded as a first-line therapy for this type of haematuria. Finasteride can also be used to stop male baldness. It seems particularly effective in men aged 20-40 years; 85% of patients stopped losing hair when given Finasteride. When the treatment was stopped hair loss continued, thus therapy may have to be "lifelong".
Publication Types:
Review
Review, Tutorial
G
Guest
Guest
For those who are worried about gyno read this
Perhaps this is the way to go vs. Propecia See below from Dr. Pickhart
Is Copper Better Than Propecia® for Blocking DHT Production?
Recent research indicates that the DHT that harms hair follicles comes from the the skin's sebocytes and sweat glands (sebaceous glands). (Chen et al 1996) 5 alpha-Reductase, the enzyme system that converts testosterone into DHT occurs in two enzyme forms. The type 1 represents the 'cutaneous type'; it is located primarily in the skin's sebocytes but also in epidermal and follicular keratinocytes, dermal papilla cells and sweat glands as well as in fibroblasts. The type 2 is located mainly in the seminal vesicles, prostate and in the inner root sheath of the hair follicle.
Propecia® (Finasteride), which has a higher affinity for the type 2 form, is best suited for for controlling prostate enlargement. It also must be administered by pills that spread the drug throughout the body.
Copper ion in the skin is more effective in inhibiting the type 1 form which is primarily producing the DHT that damages follicles and can be administered locally to the skin. Sugimoto et al (Sugimito 1995) found that copper ion is a potent inhibitor of 5-alpha reductase, inhibiting both types of 5-alpha reductase (both type 1 and type 2) that produce DHT and is the only metal to do so. Copper ion inhibits (50% reduction in activity) type 1 alpha reductase at 1.9 micromolar (0.12 micrograms copper ion per milliliter) and type 2 alpha reductase at 19.2 microM (1.2 micrograms copper ion per milliliter). No other metal has these effects.
The application of copper-peptides may provide sufficient copper ion into the hair follicle area to block DHT production in the scalp. Metabolically active copper ion (that is, copper ion free to block 5-alpha reductase) in the human body exists at about 1 microgram per milliliter in the blood and less in the skin. While the uptake of ionic copper from copper-peptides applied to the scalp is very low, human experiments by Prof. John Sorenson (University of Arkansas) have found that they can raise copper ion levels in the skin to the effective level of 1 microgram per milliliter.
Perhaps this is the way to go vs. Propecia See below from Dr. Pickhart
Is Copper Better Than Propecia® for Blocking DHT Production?
Recent research indicates that the DHT that harms hair follicles comes from the the skin's sebocytes and sweat glands (sebaceous glands). (Chen et al 1996) 5 alpha-Reductase, the enzyme system that converts testosterone into DHT occurs in two enzyme forms. The type 1 represents the 'cutaneous type'; it is located primarily in the skin's sebocytes but also in epidermal and follicular keratinocytes, dermal papilla cells and sweat glands as well as in fibroblasts. The type 2 is located mainly in the seminal vesicles, prostate and in the inner root sheath of the hair follicle.
Propecia® (Finasteride), which has a higher affinity for the type 2 form, is best suited for for controlling prostate enlargement. It also must be administered by pills that spread the drug throughout the body.
Copper ion in the skin is more effective in inhibiting the type 1 form which is primarily producing the DHT that damages follicles and can be administered locally to the skin. Sugimoto et al (Sugimito 1995) found that copper ion is a potent inhibitor of 5-alpha reductase, inhibiting both types of 5-alpha reductase (both type 1 and type 2) that produce DHT and is the only metal to do so. Copper ion inhibits (50% reduction in activity) type 1 alpha reductase at 1.9 micromolar (0.12 micrograms copper ion per milliliter) and type 2 alpha reductase at 19.2 microM (1.2 micrograms copper ion per milliliter). No other metal has these effects.
The application of copper-peptides may provide sufficient copper ion into the hair follicle area to block DHT production in the scalp. Metabolically active copper ion (that is, copper ion free to block 5-alpha reductase) in the human body exists at about 1 microgram per milliliter in the blood and less in the skin. While the uptake of ionic copper from copper-peptides applied to the scalp is very low, human experiments by Prof. John Sorenson (University of Arkansas) have found that they can raise copper ion levels in the skin to the effective level of 1 microgram per milliliter.
Hey
Bruce...What?!! This article just said that 5-AR Type 1 is the enzyme that damages hair follicles, but finestride works to eliminate Type 2....what's up with that?
"Copper ion in the skin is more effective in inhibiting the type 1 form which is primarily producing the DHT that damages follicles and can be administered locally to the skin."
Bruce...What?!! This article just said that 5-AR Type 1 is the enzyme that damages hair follicles, but finestride works to eliminate Type 2....what's up with that?
"Copper ion in the skin is more effective in inhibiting the type 1 form which is primarily producing the DHT that damages follicles and can be administered locally to the skin."