I can use distilled water and lactoferrin. Is the elder brother who uses lactoferrin recovered? Where is a step back?lol You dislike my post after I help you out. I should've told you to use ethanol as a vehicle so you'd kill it.
Pgd2 ?how Can I Do It?help Me Help Me
- Thread starter killDHT
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- cetirizine pgd2
I can use distilled water and lactoferrin. Is the elder brother who uses lactoferrin recovered? Where is a step back?lol You dislike my post after I help you out. I should've told you to use ethanol as a vehicle so you'd kill it.
Why do you want to lie? He's improving, but you say he's retreating. You're wrong.
The second picture is obviously better than the first one. Even if you don't see it, it has been used for more than 8 months.
You can see in his introduction that he is partial lactoferrin.
That post is full of lactoferrin for external useThat post is full of lactoferrin for external use
This person, using lactoferrin external solution, the second picture is 2020, the first picture is 2019, he also did not use finasteride or dutasteride. Isn't that progress?Sorry, I meant the second picture looks better at first glance. He said the second picture is the before pic.
Anyways, good luck to you. I just think you're wasting your time.
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the wnt pathway is stimulated before the hedgehog pathway. In another embodiment, the two pathways are stimulated in an overlapping fashion. In another embodiment, the two pathways are stimulated simultaneously.
At 25 and 45 days after wound induction, wound sites contained new hairs (FIG. 11, left and right panels, respectively). New hairs appeared to lack pigmentation, except when the wnt pathway was inhibited, using Dkk-1 (Dickkopf-1) during the first nine days after wounding (see Example 10).
In other experiments, continued expression of Dkk1 after the 9-day period inhibited formation of new HF.
21 day-old mice were wounded as described in previous Examples. Starting from day 11 after wounding, a time point corresponding to the point at which the wound had recently reepithelialized, 10 μL of 1 μg/ml EGF was injected into the wound bed. EGF was injected once per day after this point for a total of 5 days. Three days later, the skin was collected, and whole-mount EDIHN assays were performed. EGF prevented HF formation as assessed by gross morphology. In addition, whole mounts of control and treated skin were analyzed with anti-K17 antibody immunostaining. All mice injected with EGF (n=4) exhibited no new HF formation. while control mice (n=2) had many new HF, as expected.
https://patents.justia.com/patent/20200101000
At 25 and 45 days after wound induction, wound sites contained new hairs (FIG. 11, left and right panels, respectively). New hairs appeared to lack pigmentation, except when the wnt pathway was inhibited, using Dkk-1 (Dickkopf-1) during the first nine days after wounding (see Example 10).
In other experiments, continued expression of Dkk1 after the 9-day period inhibited formation of new HF.
21 day-old mice were wounded as described in previous Examples. Starting from day 11 after wounding, a time point corresponding to the point at which the wound had recently reepithelialized, 10 μL of 1 μg/ml EGF was injected into the wound bed. EGF was injected once per day after this point for a total of 5 days. Three days later, the skin was collected, and whole-mount EDIHN assays were performed. EGF prevented HF formation as assessed by gross morphology. In addition, whole mounts of control and treated skin were analyzed with anti-K17 antibody immunostaining. All mice injected with EGF (n=4) exhibited no new HF formation. while control mice (n=2) had many new HF, as expected.
https://patents.justia.com/patent/20200101000
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What good EGF inhibitors can be applied to the scalp or are injections preferred?
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PIGMENTATION
Why do you think that would be the case?
Most of my Wounding + MIN induced regrowth looks unpigmented indeed
(though it probably is more overgrown vellus rather than neogenesis):
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Are there any studies showing some experiment on dosage?
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I can't get my hands on it.
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I was asking about WAY-262611. Is there a dosage for that? Curious as you are.
How do you séquence these?
Wounding (follica method every other week) >
shh agonist PWD1 > SAG 0.15% twice a month?
EGFR inhibitor PWD1-PWD15 > what is it ???
DiKK iinhibitorfrom PWD10 (WAY-262611) > ???
PGE2 from PWD10 > ???
17β-Estradiol 6mg day >
anti-androgens (e.g. dutasteride, RU, spironolactone, bica) > Bica 50mg daily RU 5%
minoxidil 5mg twice a day > oral only
Stenox WAY-316606 > 1% daily
How do you séquence these?
Wounding (follica method every other week) >
shh agonist PWD1 > SAG 0.15% twice a month?
EGFR inhibitor PWD1-PWD15 > what is it ???
DiKK iinhibitorfrom PWD10 (WAY-262611) > ???
PGE2 from PWD10 > ???
17β-Estradiol 6mg day >
anti-androgens (e.g. dutasteride, RU, spironolactone, bica) > Bica 50mg daily RU 5%
minoxidil 5mg twice a day > oral only
Stenox WAY-316606 > 1% daily
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SAG just after wounding?
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ERa Vs ERb
Considering your expertise & join date, you are probably aware of this -
but just in case you are not, @IdealForehead has written a solid analysis thread covering most of your questions:
- the Estrogen receptors,
- their role in Hairloss / (Re)growth,
- the ones we should therefore Agonize (i.e. ERb), and
- the Agonists he recommends (i.e. Estriol, S-Equol etc.);
You can check it all out here:
https://www.hairlosstalk.com/intera...trogen-promote-and-hinder-hair-growth.113077/
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KETOCONAZOLE
Beside the point you mention (which I didn't check), I believe the 2% Shampoo should be part of every regimen, because:
- NO EFFORT - just replace your regular Shampoo;
- IT WORKS - plenty of study-based Evidence (and anecdotes):
https://www.hairlosstalk.com/intera...eam-2-vs-nizoral-shampoo.105066/#post-1471668
https://www.hairlosstalk.com/intera...ike-prince-william.111390/page-3#post-1612291
Edit: https://www.hairlosstalk.com/intera...ctive-oral-anti-androgen.109041/#post-1561125
- IT'S SAFE - doesn't go Systemic:
https://www.hairlosstalk.com/intera...-cyp17a-cyp17a1-thoughts.110402/#post-1591332
I would add that - to the contrary of what most recommend - for best results it should probably be used DAILY and together with the 2% Keto CREAM (I do so), because
"...3x/week is for fungal infections. For hair you want daily. Use conditioner if your hair/scalp gets too dry. Obviously the cream will be much more effective though since it's on your scalp 24/7."
https://www.hairlosstalk.com/intera...ike-prince-william.111390/page-4#post-1612314
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ER-beta is more prominent in human hair follicles (see especially Fig. 3), so while I don't know for sure I'd lean toward beta being more important.
As for erteberel, was hair regrowth ever reported as a side effect in trials?
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What's a good source for shampoo & cream 2%? The stuff on amzn is 1% in the US
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