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My ex-wife claims that I am an embarrassment to her and the children. But the way that I see it, I am adorable.
Hair is not Life but it's Pretty Damn Close; HRT and Pictorial Posts Prove it.
- Thread starter JaneyElizabeth
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why did you decide to transition?
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I look so young here; And very close to androgynous but we can see just how powerful a feminizing effect that we have from framing our faces. It puts Janey over the top in my opinion and that's why it favors me plus it's nicer in terms of volume and body style which we forget when we speak only of coverage. Gergely as your hair gets longer, I predict that it outs you more and more as it provides framing but virtually all MtF's would be thrilled to have your face and hair effects. Goddess loves you, this I know....
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Clarification about Monotherapy with Either CPA, MPA, spironolactone or the 'Mides and Safety
from shrouded_reflection via /r/AskMtFHRT sent 6 minutes ago
Loss of bone density is due to low estrogen levels (or in the case of men low testosterone levels, as testosterone is aromatized into estradiol), so as long as whatever your medication is doesn't result in low functional levels of both of those hormones you don't have any issues with density loss. In the short term it doesn't matter too much, and the body can bounce back from small periods of loss, but in the long run you need to maintain adequate hormone levels or risk early onset osteoporosis and other similar conditions.
Finasteride only blocks DHT production, and since DHT can't be aromatized to estradiol then it shouldn't have any impact on T or E levels, so no change in bone density. I would assume a similar effect with Dutasteride, but it's a drug I'm less familiar with.
For progestins such as CPA, MPA, you are downregulating the hypothalamic–pituitary–gonadal loop, which results in reduced T and E production. Supplementation with hormones is required in the long run.
Bicalutamide does seem to have slight estrogenic effects, however it isn't sufficient to offset the loss of estrogen via aromatization when it comes to bone density. spironolactone also falls into this category. Either way, estradiol supplementation is needed if your body doesn't already produce it in significant quantities.
Raloxifene can be used as an estrogen replacement in conjunction with an AA if for some reason the full effects of estrogen are not desired (raloxifene does not activate estrogen receptors in breast tissue). However it is not as effective at maintaining bone density and has a number of potentially serious side effects in the long run, so should be considered only a method of last resort, such as for non binary individuals who can't get a mastectomy.
from shrouded_reflection via /r/AskMtFHRT sent 6 minutes ago
Loss of bone density is due to low estrogen levels (or in the case of men low testosterone levels, as testosterone is aromatized into estradiol), so as long as whatever your medication is doesn't result in low functional levels of both of those hormones you don't have any issues with density loss. In the short term it doesn't matter too much, and the body can bounce back from small periods of loss, but in the long run you need to maintain adequate hormone levels or risk early onset osteoporosis and other similar conditions.
Finasteride only blocks DHT production, and since DHT can't be aromatized to estradiol then it shouldn't have any impact on T or E levels, so no change in bone density. I would assume a similar effect with Dutasteride, but it's a drug I'm less familiar with.
For progestins such as CPA, MPA, you are downregulating the hypothalamic–pituitary–gonadal loop, which results in reduced T and E production. Supplementation with hormones is required in the long run.
Bicalutamide does seem to have slight estrogenic effects, however it isn't sufficient to offset the loss of estrogen via aromatization when it comes to bone density. spironolactone also falls into this category. Either way, estradiol supplementation is needed if your body doesn't already produce it in significant quantities.
Raloxifene can be used as an estrogen replacement in conjunction with an AA if for some reason the full effects of estrogen are not desired (raloxifene does not activate estrogen receptors in breast tissue). However it is not as effective at maintaining bone density and has a number of potentially serious side effects in the long run, so should be considered only a method of last resort, such as for non binary individuals who can't get a mastectomy.
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Watching Point Break for the first time as it should be a good hair movie....
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Hey, Janey. Was wondering if you could take a look at my hair. Been on finasteride for 9 months, feel like my hairline has still gone back slightly. Only 19 and wanna keep as much as possible.
recent
4-5 months in
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4-5 months in
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You might be the one in one hundred that actually only has a maturing hair line as almost everyone loses a little in the corners and in front of the ears. That's where my substantial growth has been along with the crown filling in completely.
I would try min at two percent in addition. Why only 2 percent? My feeling is that starting lower helps prevent sheds and right now you mostly need maintenance as your hair quality looks fantastic. Most with male pattern baldness see a quick rise in frizziness which you don't have at all.
I would also encourage that you use Keto plus a blue shampoo for yeast inflammation and I bet you will maintain your more or less pubertal locks fine although eventually everyone tends to get what I call the mullet effect after 40 say. I don't even think you need to derma-roll but that's great for your face too, so I use it on both. Some XY's who are desperate, I try to mention potential HRT protocals.
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Thank you very much I really appreciate you!
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Check back with me and update me and you can follow my recovery from cueball status 18 months ago.
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You know what else? You have a window's peak and that is highly protective of a person's hairline for life with folks like Keanu and others maintaining well. Yours is perfect, protective but not like Eddie Munster.
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You sort of have a widow's peak too. That can be predictive of maintenance as I watch hairlines. You just don't want a low one because it looks vampire like but yours is perfect.
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The first too Imgur links are from last week, the other is from 3-4-5-6-7 months of finasteride. Feel like it’s been worse, what do you think I should do?
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I wanna add that I have a brother with the same widows peak hairline who has thinned a good amount in the temples, he’s 3 years older than me. Father has amazing hair, uncles on moms side not really but at that point it’s a dice roll.
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Which one?
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RU? Maybe?? I'm not really well informed about the topical route.
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Some people use topical spironolactone, but it doesn't seem to be worth it.
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What do you think of my hair?